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SCHAFER AUTISM REPORT                "Healing Autism:

                             No Finer a Cause on the Planet" ________________________________________________________________

Monday, November 17, 2003                        Vol. 7  No. 230

 

 

 --- > PROMOTE YOUR MEETINGS, CHAPTER OR CONFERENCE

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       In the Largest, Widest Read "The Autism Calendar"tm

       http://home.sprynet.com/~schafer/frm/calendar-form.htm

 --- > NOTE CALENDAR DEADLINE ** Nov 24 ** FOR DEC UPDATE

 

 

    RESEARCH

   * M.I.N.D. Institute’s Aggressive Pursuit of Possible Autism-Vaccine Ties

   * Use of Combinatorial Peptide Libraries to Identify Antigenic Epitopes

     in Children with Autism

   * Neurodevelopmental Damage Following Exposure to Thimerosal-Based

     Vaccines in Mice Vulnerable to Autoimmune Disease

   * Adverse Reaction to Vaccination- Evaluation of Clinical Immunization Safety

   * The Role of Maternal Immunity in Susceptibility to Measles Virus

     Infection and Disease

   * MMR Vaccine and Autism: A Prospective Study

 

   * Autism Summit Conference

 

    MEDIA ALERT

   * Autism Summit Conference LIVE via Web Cast

   * Attendees Take Notes!

   * Conference Agenda

   * Savantism Featured in Wired Magazine

 

    CARE

   * How Doctors Turned Asperger’s Woman Into A Twitching, Bloated Wreck

   * Shut Out Of Taser Review: Cops Won't Interview Autistic Victim or Dad

   * Former Wisconsin School Employee Accused of Child Abuse

   * Adults with Autism: Study Uses Mobile Technology To Try To

     Connect With Autistic

 

    EDUCATION

   * Award for UK Pioneer In Autistic Education

 

 

RESEARCH

 

M.I.N.D. Institute’s Aggressive Pursuit of Possible Autism-Vaccine Ties Cause célèbre d’ jour?  Cause infâme d’ jour?

 

      [Thanks to ValeriesList and Rick Rollens.]

 

      The M.I.N.D. Institute Research Program is avidly investigating potential links between vaccines and autism. During 2001 the program funded five outstanding research proposals following a nationally publicized Request for Proposals. Scientists at the University of California, Davis, the University of California, Irvine, the University of Minnesota, the Fox Chase Cancer Center in Philadelphia, and most recently, the Kennedy-Krieger Institute and Johns Hopkins University in Baltimore, will conduct studies on a broad spectrum of related topics, including: immune responses in autistic versus non-autistic children against measles, mumps and rubella proteins and determination of whether autistic children form antibodies that cross react with brain components such as receptors leading to autistic symptoms.

      A potential animal model of autism induced by early and repeated exposure to agents that are present in certain vaccines.

      The possibility that some children with “regressive” autism have an aberrant immune response that may lead to food allergies.

      The relationship between a mother's immune response and that of her newborn, using a novel mouse model.

      The developmental, gastrointestinal, sensory and other characteristics of infant siblings of children with autism pre- and post-vaccination. Together these studies critically test the possibility that the immune systems of certain, susceptible individuals react differently to, or are differentially affected by, vaccines or vaccine components which leads to alterations of brain function and the development of autism. While many organizations have been hesitant to support research on this question, the M.I.N.D. Institute Research Program is committed to helping to resolve the issue of whether vaccines may contribute to the development of autism. If such a link is found, the research could lead to the development of safer vaccine alternatives or vaccination protocols. If no link is found, parents could be reassured about the safety of childhood vaccines.

* *

 

Use of Combinatorial Peptide Libraries to Identify Antigenic Epitopes in Children with Autism

      PI: M. Eric Gershwin, University of California, Davis Collaborators: Kit S. Lam, Judy A. Van de Water, and Beth Goodlin-Jones, University of California, Davis

 

      The pathogenesis of autism is unknown. However, there have been significant concerns that its incidence is increasing. A recent report has suggested that there is a possible causal relationship between autism and vaccination. Vaccinations provide important protection for many critical diseases. Within the scientific community, there has been interest in the development of immunopathology following some viral vaccines.

      Indeed, reduction of platelets, some forms of arthritis, and some neurologic complications have been reported following rubella and influenza vaccines, amongst others. If vaccines are incriminated in the development of autism, then we believe that a critical mechanism may be that the immune response of autistic children reacts differently to some forms of the viral vaccine, than does that of control non-autistic children. This immune response of the autistic children might cross react with neuroreceptors or other central nervous system components critical for the appearance of the clinical features of autism.

      We will take advantage of a unique high throughput technology that allows us to define the immune response of children using either sera or spinal fluid. This technology further permits the “mapping” of the immune response so that the “signature” of each child against the vaccine can be defined. We hope to determine if the signature of autistic children against measles, mumps and rubella proteins within the vaccines differs from that of control children.

      We will further use this unique technology to create an extensive library of random peptides on a microbead system to determine if we can define a unique “signature” in the spinal fluid of autistic children. The success of our project may lead to the resynthesis of the important epitopes that we have defined. These epitopes, if they cross-react with neuroreceptors, may indeed be coined mimotopes. By searching various gene banks for the sequences we identify, we may be able to determine whether any proteins, not just from m easles, mumps and rubella, may be associated with autism. Indeed, this proposal offers the potential to create a diagnostic chip that can be used to screen patients to vigorously define associated risk factors.

      This proposal may be considered a high risk, but extremely high yield, investigative effort that combines the skills of an investigative immunologist, a combinatorial chemist, and a virologist with clinical specialization in autism.

* *

 

Neurodevelopmental Damage Following Exposure to Thimerosal-Based Vaccines in Mice Vulnerable to Autoimmune Disease

      PI: Mady Hornig, University of California, Irvine Collaborator: W. Ian Lipkin, University of California, Irvine, and Columbia University

      Dramatic increases in reported diagnoses of autism, a chronic and disabling neuropsychiatric disorder characterized by abnormalities in social interaction, learning, and behavior, have occurred state-, nation- and worldwide over the past decade. Although the basis of the disorder is unknown, genetic, immune and infectious factors are proposed to be important. One hypothesis is that pre- or early postnatal infection and/or exposure to toxins or vaccines results in aberrant brain development.

      his project explores an animal model of autism based on early exposure to toxins present in certain vaccines (ethylmercury present in thimerosal

additive) with or without concurrent immune challenge (polyvalent vaccinal

antigens) by repetitive bolus administration that mimics the early childhood immunization schedule. Similarities between sequelae of organic mercury exposure and autism include stereotypic behaviors; abnormal responses to novel environment; abnormal taste preferences; disturbed spatial learning; growth delay; hippocampal, cerebellar, and amygdalar pathology; autoantibodies to brain components, and increased frequency in males. Using this model, we will probe the mechanisms by which early toxic and immune disruptions alter brain architecture and function.

      Specific aims include:

      1) characterize the behavioral, cognitive, and motoric deficits associated with early thimerosal/polyvalent vaccine exposure;

      2) map the neurodevelopmental histopathology using stereologic methods;

      3) evaluate the role of cytokines, apoptosis-related products, metallothioneins (a group of proteins that bind mercury), glutathione, and other soluble factors as potential mediators of brain damage following toxic and/or immune challenge; and

      4) establish the biological basis for neonatal identification of vulnerability to neural damage following toxic and/or immune challenge through assessment of baseline levels of cytokines, metallothioneins, and other soluble factors.

* *

 

Adverse Reaction to Vaccination- Evaluation of Clinical Immunization Safety

 

      PI: Harumi Jyonouchi, University of Minnesota

      The aggressive infant immunization program greatly reduced mortality and morbidity of common infectious diseases in childhood. However, parents of “regression autism” patients often describe that development regression and autistic behavior started following immunization. Nevertheless, a role of childhood immunization in regression autism is highly controversial and poorly understood. Vaccines are designed to induce immune responses against key components of pathogens to obtain protective levels of antibodies (Abs) and sustained immune memory.

      The immune system requires additional signals to respond to antigens (Ag). In the case of infection, microbial products, such as endotoxins, bacteria/viral DNA and RNA and those released by damaged tissues, provide such additional signals that are often called 'danger' signals. The body reacts to the 'danger' signals independent of Ag, rapidly mounting non-specific, proinflammatory responses (innate immune responses). Proinflammatory cytokines produced in innate immune responses include tumor-necrosis factor-a (TNF-a), interleukin-1 (IL-1), and IL-6. They also affect the brain and induce stress responses such as fever, chills, joint aches, etc. Since infants are poorly responsive to Ags, infant vaccines are designed to boost these innate immune responses to obtain protective immunity using additional components (adjuvants).

      However, excessive innate immune responses can cause aberrant immune responses and may lead to autoimmunity. The results of our pilot study indicate that some children with regression autism demonstrated excessive production of proinflammatory cytokines with endotoxin, indicating aberrant innate immune responses. In this study, we hypothesize that adjuvants in infant vaccines may cause excessive/aberrant innate immunity and affect resultant Ag-specific immune responses (adaptive immune responses) in susceptible individuals. This may result in immune reactivity to food antigens (Ags) and even autoimmunity involving the central nervous system. The specific aim of this study is to characterize innate and adaptive immune responses in patients with adverse reactions to immunization, by measuring production of various soluble mediators in autistic and control children when peripheral blood cells are stimulated with various agents including adjuvants and food Ags.

      We expect that such immunological characterization will help us subgroup patients with regression autism. Clinical responses assessed by neuropsychiatric measures will be compared to the results obtained. Our long-term objective is to develop measures to identify individuals at high risk for severe adverse reaction to immunization and regression autism at an early age on the basis of immunological and genetic characterization. Such measures will be extremely important for prevention of regression autism. Our approach should also help predict the efficacy of certain anti-inflammatory treatments in subset(s) of regression of autism patients.

* *

 

The Role of Maternal Immunity in Susceptibility to Measles Virus Infection and Disease

      PI: Glenn F. Rall, Fox Chase Cancer Center

      Recent reports have fueled a debate within both medical and parent communities concerning the safety of childhood vaccines and their possible link to autistic spectrum disorders (ASD). However, such a link, if it exists, is not well defined. Nevertheless, because of widespread media coverage of this controversy, many parents have considered not vaccinating their children, placing in peril the widespread vaccine coverage that the United States has maintained for decades. It is therefore imperative that this issue be resolved, either leading to the development of safer vaccine alternatives, or reassuring parents of the safety of childhood vaccines.

      Current efforts to define the basis of autism have generally been restricted to the affected child, although it is increasingly apparent that the development of a child's immune response is strongly influenced by maternal immunity, both during gestation and postnatally through the transfer of antibodies in breast milk.

      Thus, if vaccines are involved in the etiology of autism, the possibility that altered immunity may contribute to increased risk seems tenable. In this proposal we explore, in an animal model of infection and immunity, how previous maternal antigen exposure may either facilitate viral clearance or lead to immunological tolerance in newborns. Our experiments will predominately take advantage of a novel transgenic mouse model of measles virus infection.

      These mice have been genetically engineered to express the measles virus receptor, CD46, specifically targeted to neurons, rendering them susceptible to measles virus infection and CNS disease. The development of disease in this animal model, however, is age-dependent: while measles virus-infected adult mice mount a protective immune response which clears the virus and affords protection, neonates cannot resolve the infection, despite a similar induction of a robust immune response. Interestingly, newborn mice weaned on mothers that were previously exposed to measles were protected from CNS disease, suggesting that the maternal exposure history afforded protection to her offspring. In Aim 1 of this project, we describe experiments to characterize the basis of such maternally-derived protection.

      We will test the hypothesis that maternal antibodies are transferred to developing embryos to afford resistance against subsequent, post-natal measles virus challenge. In performing these pilot experiments, we also assessed the response of pups that were weaned on mothers that were exposed to measles virus long before conception. These pups were not protected, suggesting that maternal protection was transient. Moreover, pups born to these mothers not only developed CNS disease, but showed no evidence of immune induction, suggesting that the previous maternal exposure tolerized her offspring to subsequent viral challenge.

      In Aim 2, we describe experiments to address the basis for this apparent tolerance, and will establish when during development such tolerance is conferred. The overall theme of this work is to explore the hypothesis that the immune response of a newborn mouse is directly influenced by the exposure history of its mother. These data will contribute to an understanding of how vaccines in some children may establish undetected, smoldering infections that may initiate a cascade of events leading to neurodevelopmental damage.

* *

 

MMR Vaccine and Autism: A Prospective Study PI: Rebecca Landa, Kennedy Krieger Institute and Johns Hopkins University

      Abstract not yet available

 

 

 

 

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* * *

 

Autism Summit Conference

 

http://www.eurekalert.org/pub_releases/2003-11/niom-asc111403.php

 

      A national conference focusing on the Federal government's role in biomedical autism research, early screening and diagnosis, and improving access to autism services will be held November 19-20, 2003, at the Washington Convention Center in Washington, DC. The summit will provide a public forum to disseminate, evaluate, and integrate the latest science-based autism information among Federal, academic, and community participants.

      A major goal of the summit is to develop an information exchange among the autism community, experts in specific areas, and Federal agencies that advance autism research and services. Another goal is to foster partnerships among these groups.

      The event, “ The Autism Summit Conference: Developing a National Agenda,” will expand the work of the Federal Interagency Autism Coordinating Committee (IACC), formed in response to the Children's Health Act of 2000. The IACC serves to enhance coordination and effectiveness of autism research and service activities across the Federal government and with public stakeholders.

      The U.S. Department of Health and Human Services and the U.S. Department of Education are conference cosponsors. Interagency sponsors from the National Institutes of Health include: The National Institute of Mental Health (NIMH), the National Institute of Child Health and Human Development (NICHD), the National Institute of Neurological Disorders and Stroke (NINDS), the National Institute on Deafness and Other Communication Disorders (NIDCD), and the National Institute of Environmental Health Sciences (NIEHS), as well as the Centers for Disease Control and Prevention (CDC), Health Resources and Services Administration (HRSA) and Substance Abuse and Mental Health Administration (SAMHSA).

      The summit's biomedical component will address autism research on diagnosis, causes, neuroscience, and treatment. Programs will build on the work of an expert panel of scientists that responded to a congressional request for a “roadmap” to identify and advance high-priority research goals. Federal officials, researchers, and community members will discuss such topics as genetics, epidemiology, and early intervention.

      The second major theme, implementing autism screening and diagnosis, will review existing screening instruments and implementing practices in the community. Relevant research and current clinical practices will be discussed.

      The third theme is integrating autism services throughout the lifespan. Topics will deal with issues for those living with autism, including fragmented services provided by educational and other systems. These subjects are similar to those raised by the President's New Freedom Commission for Mental Health.

      The summit will provide an opportunity to highlight the Federal government's interest in autism and mobilize resources to confront this recognized public health problem. It will begin to establish a national agenda of research and service goals to be achieved through advances by federal, academic, voluntary, and other public organizations.

      The conference was planned collaboratively to represent diverse interests and perspectives. Public officials, as well as major researchers and practitioners in the autism field, will be involved in all aspects of the summit.

* *

 

MEDIA ALERT

 

Autism Summit Conference LIVE via Web Cast

 

      You can view the conference LIVE or archived, via web cast, by following instructions on this link: http://www.tvworldwide.com/events/nimh/031119 .

      All general sessions will be web cast, as will one of the breakout groups for each session.  The other breakout groups will be available for archived viewing.

      In addition, you will find updated information on this conference, including the agenda and reports following the conference, on the following

website: http://www.nimh.nih.gov/autismiacc/events.cfm

* *

 

Attendees Take Notes!

 

      If you plan to attend the Summit Conference in Washington DC this week in person, or you plan to view most of it via the web cast, we would like your notes.  We are not looking for any particular slant or spin, as we intend to represent a diverse volunteer range of impressions.  Nor do we necessarily seek expert scientific review of the discussed research, or suggested research, although we would welcome such treatment.

      Here are some suggested high lights to look for:  how well are the issues of autism causation represented?  Who is promoting clinical research and who isn’t?  Do you think your interests in autism research, no matter what that may be, is being adequately represented?  Do you think your political interests as a person with family autism is being adequately represented?  What or who, if anything or anyone, is missing from this summit?

      If you would like to participate in this “community pool of reporters” , contact the editor at edit@doitnow.com. Autism awareness starts with you.

* *

 

Conference Agenda

 

http://www.nimh.nih.gov/autismiacc/events.cfm

 

Wednesday, November 19

   Master of Ceremonies: Tom Insel, M.D., NIMH

 

 9:00-9:15 am   Welcome and Introductions by Tommy Thompson Secretary of

Health and Human Services

 9:15-9:25 am   Remarks from Secretary by Dr. Rod Paige Secretary of

Education

 9:25-9:30 am   Remarks from Noel Dempsey, T.D., Minister for Education and

Science, Ireland

 9:30-10:00 am   Presentation: The Face of Autism, Jon Shestack

10:00-10:15 am   Remarks from Elias Zerhouni, M.D., NIH

10:35-10:50 am   Remarks from Congressman Henry Waxman, U.S. House of

Representatives

10:50-11:05 am   Remarks from Congressman Dan Burton, U.S. House of

Representatives

11:05-11:20 am   Remarks from Congressman Dave Weldon, U.S. House of

Representatives Public/Private Partnerships Introductions by Tom Insel, M.D., Story Landis, Ph.D., & Duane Alexander, M.D.

11:20-11:30 am   CAN Initiatives for Data Sharing, Jon Shestack, Cure Autism

Now, Steve Moldin, Ph.D., NIMH

11:30-11:40 am   ASA/CDC partnership: Autism Awareness, Campaign José

Cordero, M.D., MPH, CDC, Robert Beck, ASA

11:40-11:50 am   NICHD/NAAR High RiskfBaby Sibling Autism Research

Collaboration, Duane Alexander, M.D., NICHD

11:50-12:00 am   NAAR Autism Genome Project Prisca Chen Marvin, National

Alliance for Autism Research

 

1:00-4:30 pm

 

THEME 1: Services for Individuals with Autism Across the Lifespan

    1:00-1:30 pm Welcoming Remarks  Robert H. Pasternack, Ph.D., Assistant Secretary, Office of Special Education and Rehabilitative Services, U.S. Department of Education

    1:30-3:00 pm Concurrent Sessions

Session A: Early Childhood/Birth to Five, Moderator: Merle McPherson, M.D., Health Resources and Services Administration Adrian D. Sandler, MD, FAAP, The Graham Children’s Health Center, Sue Baker, Autism Services Consultant, Child Health Specialty Clinic, Juliann Woods, Ph.D., CCC-SLP, Florida State University, Jeff Sell, Bonnie Strickland, Ph.D., Health Resources and Services, Administration

 

Session B:   School-Age

Moderator: Gail Houle, Ph.D., Office of Special Education Programs, Ilene Schwartz, Ph.D., University of Washington, Cathy Pratt, Ph.D., Indiana Institute on Disability and Community, Andrew Bauman, Robin Welsh, M.S., M.A., Esq., Department of Special Education, Calvert County, Maryland, Beth Caron, Ph.D., Office of Special Education and Rehabilitative Services

 

Session C:   Secondary/Transition

Moderator: Joan Mete-McCarthy, D.A., Office of Special Education and Rehabilitative Services, Susan E. Levy, M.D., Children’s Hospital of Philadelphia, Teresa Grossi, Ph.D. Indiana Institute on Disability and Community, Jim Ball, Ed.D., Sawtelle Learning Centers, Mayer Max, M.S., SharkaTech, Merlene Simon-Burroughs, Ph.D., Office of Special Education Programs

 

Session D:   Adults

Moderator: Lee Grossman. Stephen Shore, M.A., Anna Hundley, NARPAA, Evelyne Milorin, Kennedy Fellow, Office of Special Education, and Rehabilitative Services, Jenn Rigger, M.S., Rehabilitation Services Administration

    3:15-3:35 pm    Report-outs from concurrent sessions

    3:15-4:45 pm    Presentations from Experts in the field, Moderator: Joan

Mete-McCarthy, D.A., Office of Special

Education and Rehabilitative Services.  Eric Schopler, Ph.D., TEACCH, University of North Carolina, Chapel Hill, David Holmes, Ed.D., Eden Services, Robert Koegel, Ph.D., University of California, Santa Barbara, Stanley Greenspan, M.D., George Washington University Medical School, Ivar Lovaas, Ph.D., Lovaas Institute for Early Intervention, Lee Grossman, ASA

    4:45-5:00 pm Open Mike Session

 

Thursday, November 20

 

8:30 - 12:00 THEME 2: Early Screening and Diagnosis

8:30-9:30 am Plenary Session -Moderator. Jose Cordero, MD., MP.H, CDC, The Current State of Developmental Screening General Developmental Screening and Screening for the ASDs,  Barry Zuckerman, M.D., Boston University and Peter Mundy, Ph.D., University of Miami

10:00-12:00   Concurrent Sessions-The “How To’s”: A Tool Box for

Developmental Screening

Session A: “How to build awareness among parents, practitioners, and educators” Moderator. Lucille (Lu) Zeph, EdD., University of Maine Speakers • Nancy Wiseman, First Signs, Inc. • Katherine Lyon Daniel, Ph.D., Centers for Disease Control and Prevention Panel Discussion • Lee Grossman • Adrian Sandier, M.D., Mission Children’s Hospital • Margaret Dunlde, George Washington University Session B: “How to test - best tests and why”

Discussant: Wendy Stone, Ph.D., Vanderbilt University Speakers • Frances Page Giascoe, Ph.D., Vanderbilt and Penn State Universities • Amy Wetherby, Ph.D., Florida State University • Deborah Fein, Ph.D., University of Connecticut

 

General Discussion

Session C: “How to put into practice-best models”

Moderator: Marian Earls, MD., Guilford Child Health, Inc. Speakers • Ed Schor, M.D., The Commonwealth Fund • Jennifer Pinto-Martin, Ph.D., University of Pennsylvania Panel Discussion • Jim O’Brien, Ph.D., Agency for Children and Families, head Start Bureau • Mary M. Gottesman, Ph.D., Ohio State University • Paul Dworkin, M.D., University of Connecticut • Eric London, M.D., National Alliance for Autism Research

 

1:00-4:30 pm  THEME 3: Biomedical Research

   1:00-2:00 pm  Overview: Autism Research Matrix and Roadmap Activities, Presentation by: Tom Insel, M.D., NIMH

   2:15-4:15 pm  Concurrent Sessions

            Session 1: Genetics Research, Leaders: Steve Moldin, Ph.D., NIMH, Dan Geschwind, M.D., Ph.D. University of California at Los Angeles, Andy Shih, Ph.D, NAAR

            Session 2: Epidemiological and Environmental Research, Leaders: Cindy Lawler, Ph.D., NIEHS, Diana Schendel, PhD., NCBDDD, CDC, Grether, Ph.D., California Department of Health Services, W. Ian Lipkin, Ph.D., Columbia University, Kathleen Berry

            Session 3: Early Intervention Research Leaders: Ann Wagner, Ph.D., NIMH, Geraldine Dawson, Ph.D., University of Washington, Richard Fade, Northwest Autism Foundation

 

 

 

 

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* * *

 

Savantism Featured in Wired Magazine

 

      Diane Savage, stage mom to musical prodigy son Matt Savage reports in her latest newsletter that “Wired Magazine has written an exceptional article about Savants, focusing a great deal on Matt and his musical and mathematical abilities, for their December issue. The issue is due out next week.”  The last treatment Wired Magazine gave autism was titled “The Geek Syndrome” and presented some dubious theories.  Let’s hope they don’t title this article “Dork Savants” or some such. Diane also mentioned “Time Magazine’s current issue (November 17th) contains an article about Matt’s debut at the Blue Note in NY, which happened the same week as classical pianist Lang Lang’s debut at Carnegie Hall.” A clipping of that review by Christopher Porterfield appeared in last Monday, Nov. 10, SAR as well. -LS

* * *

 

CARE

 

How Doctors Turned Julie Into A Twitching, Bloated Wreck

 

http://www.telegraph.co.uk/news/main.jhtml?xml=/news/2003/11/16/nasp16.xml&s

Sheet=/news/2003/11/16/ixhome.html <- - address ends here.

 

      Julie Eldred used to have a good job, a long-term boyfriend and her own home. That was 17 years ago. Since then she has been reduced to a twitching, bloated, semi-catatonic wreck by prescription drugs, which numerous experts now say she should never have been given. Julie Eldred was a bridesmaid at her sister's wedding in 1986

      What Julie, now 43, actually has is Asperger's syndrome, a relatively mild form of autism that in her case was so slight that she was able to live the first 26 years of her life with few problems.

      Asperger's is not a mental illness and cannot normally be treated with drugs. Instead, people with the condition require behavioural guidance in relating to a world they often find difficult to understand.

      When Julie first exhibited signs of anxiety, a common symptom of Asperger's, and sought medical advice, she was said to be suffering from everything from an attention-seeking disorder to mental illness.

      She was locked up and put on 32 different drugs - which have ruined her health, led to her being repeatedly held in secure psychiatric institutions and prompted her to make several attempts to kill herself.

+ Article continues:

http://www.telegraph.co.uk/news/main.jhtml?xml=/news/2003/11/16/nasp16.xml&s

Sheet=/news/2003/11/16/ixhome.html <- - address ends here.

* * *

 

Shut Out Of Taser Review: Cops Won't Interview Autistic Victim Or Dad

 

      [By Jeremy Loome, Edmonton Sun.] http://www.canoe.ca/EdmontonNews/es.es-11-14-0011.html

 

      An internal review of an incident in which a young autistic man was tasered by Camrose police will likely be wrapped up by the end of next week.

      But the review won't include interviews with the young man, his father or the group home he walked away from, said Insp. Darrell Kambeitz.

      “That is certainly not part of our review at present,” said Kambeitz. “At this point we are interviewing the officers involved as part of a procedural review.”

      Kambeitz did stress, however, that the force will discuss the issue with Dale Firkus, the boy's father, should he file an official complaint. Firkus has retained a lawyer, and is exploring his options.

      Firkus told the Sun his son, Derek Firkus, after sneaking out of his group home, was tasered by police Sunday.

      Police Confirm A Man Was Tasered  Police have confirmed a young man was tasered - wherein a suspect is briefly jolted into submission with a gun that shoots 5,000 volts of electricity - during an investigation of vehicle thefts, and was found to have items from nearby cars when arrested.

      But Firkus said yesterday that even if his son had picked up items that didn't belong to him - which has happened in the past - it couldn't qualify as theft because his son wouldn't understand what he was doing.

      Firkus said police told him his son was found with someone else's keychain and an air freshener.

      Police said the tasering occurred because the man made “abnormal movements.”

      “Anyone who talked to Derek for 10 seconds would realize that he is handicapped,” said Firkus.

      “There is absolutely no reason why they would have to handle it the way they did.”

      A spokesman for the Edmonton Autism Society said anyone trained to recognize autism would have found Derek's movements familiar.

      Past-president Anita Ferri said the case highlights the need to further train police to recognize people with disabilities.

      “It is really very characteristic of autism to have difficulty communicating, and sometimes the problem is very severe,” said Ferri.

      Firkus said his son has snuck out before and he doesn't blame the group home, which provided him with 24-hour care.

* * *

 

Former Wisconsin School Employee Accused Of Child Abuse Assistant Allegedly Struck Special Education Student

 

http://www.themilwaukeechannel.com/news/2637941/detail.html

 

      Jacob F. Lee, an educational assistant, was suspended and then fired following the incident, Racine Unified spokeswoman Linda Flashinski said Thursday.

      Lee was scheduled to appear in Racine County Circuit Court Wednesday on felony child abuse charges. He faces up to six years in prison if convicted.

      Lee, of Racine, had been assigned to provide one-on-one assistance to a 10-year-old boy who suffers from autism and attention-deficit disorders, according to a criminal complaint.

      Racine, Wis. -- A former Racine Unified School District employee has been charged with child abuse after allegedly striking a special-education student during a field trip.

      The complaint said an aide saw Lee, 52, strike the boy three times in the face after the boy acted out on a bus during a field trip May 28.

* * *

 

Adults with Autism: Study Uses Mobile Technology To Try To Connect With Autistic

 

      [By Jeff Libby in Titusville, Fla.] http://www.theledger.com/apps/pbcs.dll/article?AID=/20031116/APN/311160604

 

      In a cavernous mall department store, a teenage boy pauses by the display he has been stocking with slippers to check a message on his pocket computer.

      For the few mid-morning shoppers passing by, the scene was as mundane as the Muzak remake of an old Beach Boys tune being piped through the PA system. Thousands of business people have been using the handheld devices for nearly a decade now.

      But 15-year-old David Jaehne has autism, along with other developmental disabilities, and for him the technology is brand new. He is one of six high school students participating in a University of Central Florida study to equip autistic youths with mobile technology in an effort to tap into their largely unmapped cognitive world.

      If David understands the message on his screen, it could mean leaving behind a life of closely supervised work in shelters and qualifying for a job like the one he is attempting to perform now, working with more independence in the larger community.

      “We make all these excuses why people with disabilities can't do things rather than matching their abilities to places they can be successful,” said Kim Carper, a UCF graduate student leading the study. “I'm trying to provide a vehicle so even people with the most severe disabilities can be given the opportunity to work in the community.”

      Carper is also coordinator for the Center for Autism and Related Disabilities at UCF, one of six centers in the state that provides assistance to caregivers and families of the estimated 200,000 people in Florida with developmental disabilities. Her center serves Orange, Osceola, Seminole, Lake, Sumter, Brevard and Volusia counties.

      Before Carper began her study four months ago, care providers and researchers had been bringing mobile technology to people with mental retardation, but few if any researchers had attempted to modify the technology for work with autistic people, who in most cases are considered more severely disabled, said Dan Davies, president of AbleLink Technologies in Colorado Springs, Colo.

      The roughly 16,800 people with autism in Florida and the nearly 300,000 in the nation had essentially been left behind.

      “There's been no work done with assistive technologies that I'm aware of with that population,” said Davies, who has seen modest sales of his mobile products for other developmentally disabled people double since he introduced them in 2001. “There's still more unknown about what helps people with autism than what is known.”

      There is disagreement over how much impact Carper's study could have, but most care providers and researchers in the field agree that any gains in independence and in productivity will be helpful to the self-esteem of the individuals and to the bottom line of the state, which spends close to $118 million annually to provide vocational work and oversight for disabled people.

      Carper's boss, Chad Nye, executive director of the university's Center for Autism and Related Disabilities, cautioned that with technology changing so rapidly, Carper's study should be viewed as a “snapshot” of a small test group in a tightly controlled environment.

      Despite its small scope, the study challenges long-held notions about autism and also offers potential relief to the high cost of providing close, constant supervision of severely developmentally disabled people, Nye said.

+ Article continues: Study Uses Mobile Technology To Try To Connect With

Autistic

* * *

 

EDUCATION

 

Award for UK Pioneer In Autistic Education

 

      [By Colin Birch.] http://icberkshire.icnetwork.co.uk/0100news/sloughandwindsor/content_objecti

d=13624058_method=full_siteid=50102_headline=-Award-for-pioneer-in-autistic-

education-name_page.html <- - address ends here.

 

        A woman who runs social skills workshops for college students with autism has picked up a national award.

      She is Jenny Ilic, Asperger Syndrome co-ordinator at East Berks College, who has won a National Autistic Society (NAS) Autism Inclusion Award for her pioneering work.

      Jenny and her team support more than 90 students, running social skills workshops that include discussions about body language, bullying, social interaction and anything else the students need to continue their positive college experience.

      In creating Jenny's role, and through her hard work and enthusiasm, East Berks College is helping students with AS learn life skills as well as academic subjects at a mainstream college.

      She collected the award at a ceremony at Lincoln's Inn, London, on Wednesday, from NAS president Jane Asher and Liz Blackman, MP, chairman of the All Party Parliamentary Group on Autism.

      Liz Blackman said: “Further education is a critical transition period for many students especially so if they have communication and other problems.

      “They can feel especially vulnerable in a new less intimate environment.

      “Jenny was nominated by a student with autism who wrote very movingly and positively about his own experience.

      “The workshops support many students to socialise, become more independent, express and solve their problems and gain greater confidence.”

 

_________________________________________________________________

Lenny Schafer, Editor mailto:edit@doitnow.com

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