Vaccination is known to result in a
subclinical
or mild case of the disease for which one is being vaccinated
against.
Rubella (German measles) is a disease for which
there is a widely used vaccine. (It is generally administered via
the MMR vaccine.) It is, in itself, a mild, often even subclinical
disease. The reason it is recommended/mandated is to protect
the developing fetus, which if it is exposed to the virus during a
critical period while still in the womb, can develop a serious
condition called
congenital
rubella syndrome.
There is evidence that the
rubella
vaccinated, including
pregnant
women, are getting subclinical cases of rubella, just as is the
rest of the population.
What does vaccination accomplish if all it does is result in a
subclinical case of an already mild or subclinical disease?
Moreover, what does vaccination accomplish if
previously vaccinated pregnant women not only can get subclinical
cases of rubella, but that such a cases can result in
congenital rubella syndrome (CRS)?
Unfortunately rubella vaccine immunity also
appears to
wane
over time. On the other hand,
natural
immunity to rubella is thought to generally be lifelong.
Would it not be better to expose children,
particularly non-immune teenage girls, to rubella in order to increase
the likelihood that one would be immune during pregnancy, rather than
to risk the possibility of getting rubella while pregnant? As
Dr. Hugh Paul stated in "The Control of Diseases", which was written in
1964, before formulation of the rubella vaccine, "The disease (rubella)
cannot be prevented, and in view of its very mild character, and the
possibility that it may have catastrophic effects if contracted by an
expectant mother, it is questionable if it should be prevented in
childhood and adolescence even if this were possible. It has been
suggested that female children should be deliberately exposed to
infection in order to achieve a life-long immunity from the disease and
possibly from malformation in the offspring in later life.
This idea is not an unreasonable one... Rubella does not kill, and even
complications are uncommon."
And if rubella during pregnancy is often
subclinical among the vaccinated, how diligent are we being in
evaluating newborns for congenital rubella syndrome since
serological testing is necessary to confirm CRS and often even
rubella itself?
There are a number
of symptoms associated with the condition. Two of the
possible symptoms are "developmental delay" and hearing loss, both of
which might not be noticed immediately. Are we assuming
something isn't congenital rubella syndrome simply because the mother
was vaccinated? Are we testing every infant or child who shows
"developmental delay" or hearing loss for CRS?
The textbook "Vaccines"
lists a group of "prominent
clinical findings in congenital rubella syndrome". Among them are
autism and diabetes.
Are we testing every infant and child with autism or diabetes for
CRS?
Some of these conditions, including autism and
diabetes, are even thought to be on the rise. The question was
just raised whether we might be assuming conditions cannot be related
to pregnancy-acquired rubella merely because of widespread
rubella vaccination. Are we even doing this with conditions
thought to be increasing?
Could these increases possibly even have
something to do with rubella vaccine causing more women of
child-bearing age to be vulnerable to the disease, now that they no
longer achieve natural immunity during childhood?
Are we conducting laboratory testing of every
infant or child who shows one or more of the numerous symptoms
associated with CRS? If not, isn't it possible that we have
been overestimating rubella vaccine's success in eliminating congenital
rubella syndrome? Of even more concern, might rubella vaccine be
at least partly responsible for the rising incidence of some
CRS-associated conditions?
Sandy
Mintz