Autism
The following is submitted to the MRC
review of autism:
A.
Autism & Autistic Enterocolitis
1.
It is suggested that there is a strong case to
Identify
to what extent autism has increased, and whether the increases are largely down
to a sharp rise in late-onset degenerative autism
Identify
if this late-onset autism is the novel syndrome of autistic enterocolitis
identified by the Royal Free Hospital and other researchers
Identify
key research vacuums that show promise
Properly
and independently investigate the alleged link between MMR and autism/autistic
enterocolitis, without prejudice.
B.
Increases In Autism
1.
It is now increasingly obvious
- despite certain protestations
to the contrary - that there has been a major recent growth in
autism in western countries, and possibly elsewhere:
Recent
studies suggest a sharp increase in autism. In the UK, studies by Shattock
(2000), by Scott (1999), by Kaye et al (2001) and by Fombonne et al (2001) have
respectively revealed autism at eight, ten, seven and four times their
previously-anticipated rates (the Fombonne study revealed a tenfold-higher rate
if milder conditions were included).
The
September 2000 Scottish schools census showed a 30% increase over the 1999
figure, itself a 19% increase over the 1998 figure.
A
study by Wakefield LEA pointed to an increase from 5 to 111 pupils in seven
years 1992-99.
Other
anecdotal data, such as lack of significant autistic adult population, pressure
on school places, personal experience (never met/heard of single case, 1950-88,
now have three cases living within 50 yards of home), absence of Parliamentary
debate/questions pre-1990.
US
data shows an increase from 12,000 to 53,000 in six years 1993-99. Some US
States showed increases of several hundred per cent.
2.
Is it better recognition or a genuine increase?
Some
of these increases are better recognition, but they are far too steep and in
too short a timeframe for this to be the whole explanation, or even the main
explanatory factor. This is gradually being accepted by most commentators.
3.
Is MMR Implicated?
It
has been alleged that MMR cannot be causing autism, because increases in autism
pre-date the introduction of MMR into the UK. However, this assertion is
grossly over-simplistic and almost certainly false. Autism could have already
been increasing for non-MMR reasons, with MMR then further accentuating the
increase.
Also,
doubt over the figures on autism, changes in diagnostic practice and delays in
diagnosis make reliance on figures alone as “proof” of absence of a link
extremely unwise (suggestive of wishful thinking).
C.
Autistic Enterocolitis - A Novel Syndrome
1.
Several key aspects stand out:
The
children who allegedly became autistic after MMR, also allegedly acquired acute
multiple food allergies, hyperactivity and bowel problems at or around the same
time
This
is obviously a pertinent issue for investigation, yet has been largely
disregarded by the Department of Health and many researchers/commentators.
For
many children, bowel problems are probably not formally diagnosed, even by
parents, and their importance probably not recognised, due to the child’s own
lack of speech and continuing incontinence. Some food allergy problems will
also go unrecognised, and their importance will not be appreciated.
2.
Data on these potentially-associated conditions:
Data
on these potentially-associated conditions in the GP database is likely to be
extremely poor, and this may go on to confound studies based upon interrogation
of the database.
affected
children developed normally and then dramatically regressed. This is a novel
variant of autism. In the past, many cases showed autistic/delayed features by
age one year. Autistic enterocolitis cases appear to differ markedly, with all
cases featuring normal development then degeneration well beyond this age,
suggesting a novel form of autism.
The
proportion comprising this “late-onset” autism may have increased very
significantly, but studies to date have not properly differentiated between the
two variants. There is therefore a fundamental data vacuum on this vital
aspect.
3.
Time delays between immunisation, onset of symptoms, formal diagnosis:
Crucially,
the delay between MMR and first noticed signs of autism, or first diagnosis,
appears to be considerable or even very great, far greater than hitherto
supposed.
The
forthcoming study by Spitzer et al of 276 children points to a range of delay
between MMR and first sign of autism varying from 38 days to 6.2 years. The
range from MMR to first diagnosis varies from six months to 10.3 years. These
findings, which mirror the representations of affected parents, would point to
a length of delay that would fall very far outside the standard three weeks of
safety trial follow-up.
It
would also result in the connection between MMR and degeneration continuing to
be missed, by some parents, by many paediatricians, and probably by all child
health databases.
D.
Research “Disproving” A Link Between MMR/Autism
1.
Studies that “disprove” a link with MMR:
A
large number of studies have recently been completed in the US and UK that
purport to show that there is no link between MMR and autism.
However,
some of these studies have been very poorly designed, or have tested a weak and
questionable hypothesis, such as whether autism would show up in increased
visits to GPs, and then been used to attempt to statistically “disprove” a
medical condition, without any medical examination of the children who
allegedly became autistic after MMR, or even medical examination of any
children in the studies involved.
The
length of delay in degeneration outlined above has not been understood.
All
these studies may therefore be very seriously flawed (a critique of most of
these studies is available separately on request).
E.
The Research Vacuum
1.
There has been a historic research vacuum in key areas:
Key
relevant areas of research have not been done. The most obvious action, of
funding further intensive study of the preliminary findings of the Royal Free
Hospital Inflammatory Bowel Disease Study Group, has been consistently
sidestepped (the work is being funded by the parents themselves).
The
most promising leads are not being followed up by the authorities. This in
itself may be instructive, exposing an institutional bias, based upon a pro-MMR
stance and an understandable anxiety not to undermine public confidence..
A
review by Wakefield and Montgomery of the original safety trials of MMR has
exposed that these were not adequately done. Although attempts have been made
to refute this claim, Wakefield and Montgomery’s findings basically stand
unchallenged. Very few children indeed were followed up for sufficiently long
in safety trials, and the significance of reported gastrointestinal problems
was not appreciated
The
effects of vaccines administered in combination have never been properly
researched.
2.
Thiomersal in vaccines may also play a role:
The
effects of thiomersal in concert with the effects of ileal lymphoid nodular
hyperplasia in the gut have not been researched.
A
US CDC review of thiomersal in June 2000 supported a statistically significant,
albeit weak, association between exposure to thiomersal-containing vaccines and
speech delays, attention deficit disorder and other neurodevelopmental delays.
It has also been separately suggested that there is a striking overlap between
characteristics of mercury poisoning and of autism.
F.
Vaccine Adverse Reaction Reporting
1.
Part of the problem is that vaccine adverse event statistics may
seriously misrepresent the real position.
The
vaccine adverse reaction reporting system has worked in the past for acute
post-vaccination reactions. But has it worked for very slow degenerative
reactions that have hitherto not been connected with vaccines?
It
is also known that vaccine adverse reaction reporting mechanisms are very poor.
By the admission of the UK MCA/CSM, only 10-15% of even serious adverse
reactions are thought to be reported. Attempts have been made to defend
this -
but the statistic is the MCA’s own, and they used the word “serious”.
This
unreformed system will have completely missed gradual degeneration over months
or years after MMR as a potential adverse event. The US VAERS system has
similar shortcomings.
This
has given MMR a good safety record in official statistics. This record may not
in fact be justified.
2.
A key error has been to underrate the importance of what the
parents - who know their child’s history best - are reporting:
The
parents’ reports of degeneration after vaccination are being treated as
“anecdotal”, but in fact point towards a consistent pattern.
The
importance of anecdotal accounts, or of circumstantial evidence forming a
consistent pattern, is being almost completely missed.
G.
Reviews & Investigations
1.
Belatedly, a number of agencies are realising that there may be a major
problem. This realisation is parent-led, through political pressure, rather
than safety-agency led.
Hearings
are now already under way by the Government Reform Committee of the US House of
Representatives as to why there are such significant increases in autism in the
US.
In
the UK, the Scottish Executive has launched a review, following earlier
consideration by the Health Committee of the Scottish Parliament. The Health
and Children’s Committee of the Dail, Republic of Ireland, has also recently
held hearings.
Other
reviews of the case for and against a link between MMR and autism, or to
identify future areas of research, are under way. In the US, the review is
continuing, led by the Institute of Medicine. In the UK, a review to identify
research is being undertaken by the Medical Research Council.
There
is widespread disquiet amongst parents of damaged children that reviews will
again prove inconclusive, and that this inconclusivity will be misrepresented
by the respective Government health departments as fresh “proof” of there being
no link.
There
is also concern at a lack of independence of some key participants. Some
individuals appear to be effectively sitting in judgement on their earlier
actions, or lack of action.
H.
Legal Action
Major class actions are under way in both
the UK and the US Courts.
The
UK action involves several hundred children, and is well advanced. It focuses
upon MMR and autism or other forms of damage.
The
US action is only just beginning, but a large number of cases are anticipated.
It focuses upon a mercury-based preservative, thiomersal, used in childhood
vaccines. The two issues may well prove to be interlinked..
It
is therefore essential to get to the truth as to whether autism and vaccination
are linked, and for investigation of a link to be totally independent from
bodies with a vested professional interest in the outcome. Public confidence
will not be rebuilt upon false reassurance, particularly if the children go on
to win their cases.
J.
Some Suggested Key Questions:
1.
These are taken from the UK MRC workshop list of 11th July 2001, but
with abbreviation or modification where necessary:
2.
Epidemiology working group
-
(i) Can diagnostic changes alone explain perceived increases in autism?
(ii) Would a register help monitor changes in prevalence?
(iii) Is the increased prevalence uniform across all variants of
autism?
(iv) Can autism be acquired after normal development, and if so, how?
3.
Physiology working group -
(i) What is the role of bowel disorders?
(ii) What is the role of gut disorders/”leaky” gut?
(iii) What are the linkages between the gut, the bowel and the brain?
(iv) What are the linkages between these three and autism?
(v) What evidence is there to implicate vaccines as a trigger?
(vi) What evidence is there to implicate antibiotics as a “primer”?
4.
Psychology working group -
(i) Why are sleep patterns so disturbed in autistic children?
5.
Other key questions (all working groups) -
(i) What systematic patterns (manifestation, timing, etc.) can be
deduced from the accounts of parents?
6.
Lay working group -
(i)
All the above questions.
7.
It is suggested that the above questions should form a central focus of
the current review, even if only to have to acknowledge that answers are not
known. Uncertainty needs to be recognised as a valid intellectual position.
K.
Other Points
1.
The confusion between the position “there is an absence of evidence” and
the position “an absence of evidence equates to proof against” needs to end.
2.
Suspected causative factors should only be eliminated at this early
stage if there is conclusive and unambiguous proof of evidence of no effect.
Potential suspects such as vaccines should therefore not be seen as “innocent
until proven guilty”, particularly in the absence of convincing causal studies.
3.
It should not be assumed that parents who question MMR, or who are
taking legal action over MMR, are “anti-vaccine” campaigners. All the parents
of children believed to have been damaged by MMR took their children to be
vaccinated.
David Thrower
01925-264156
Email David@ThrowerWarrington.freeserve.co.uk
16th July 2001
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