http://www.whale.to/vaccines/hib.html
HIB
VACCINE
"Classen's data and other published data
indicates the following vaccines are associated with an increased risk of
diabetes (increased risk): hepatitis B (50%), hemophilus (25%), tetanus (20%),
diphtheria (9%), pertussis (25%), mumps- rubella (23%). These findings
are supported by a case control study performed in Europe. The
cumulative effect of all these vaccines on diabetes is tremendous."--PRNewswire
The
Perilous Haemophilus or is it....pneumonia By Hilary Butler
What
is Hib disease?--Greg Beattie
HAEMOPHILUS INFLUENZAE-B
VACCINATION PROGRAMS HIGHLY EFFECTIVE, BUT HOW SAFE?
CHILDHOOD IMMUNISATION AND
DIABETES MELLITUS
Can Hib Vaccine Cause Asthma?-----
Heidi White
http://webpages.netlink.co.nz/~ias/hib.htm
Human data http://vaccines.net/hemophil.htm
Mouse data http://vaccines.net/newpage41.htm
Rat data http://vaccines.net/newpage6.htm
"hib is not a disease but a type of bacteria---defined by lab
test....so hard to see if there is any disease decline.......Primary motive was
to combat "invasive bacterial infections", but no evidence this has
been achieved. 3 major types of "ibi"--hib, pneumococcal, and
meningococcal. ..decline in hib infections appears to have been accompanied
by an increase in the other two.... there appears to be no evidence of a
decrease in invasive bacterial disease overall.... in aus notifications of
meningococcal disease in 1995 was highest since 1979...this rise occurred in
parallel with the fall in hib disease, so what savings in illness have there
been?....so, there seems to be no demonstration savings in illness in children,
on top of this there seems to be an association between dpt vacc and invasive
hib disease."--Greg
Beattie
"I have published many articles linking vaccines and diabetes. In
one study, a clinical trial on the hemophilus vaccine, I showed that the
risk of the vaccine exceeds the benefit. This is published by the
British medical Journal."--Bart Classen
National Vaccine Injury Compensation Program Vaccine Injury Table http://www.hrsa.dhhs.gov/bhpr/vicp/table.htm#
Schreurs AJ, Nijkamp FP. Bronchial hyperreactivity to histamine
induced by Haemophilus influenzae vaccination.
Bronchial hyperreactivity to histamine 4
days following vaccination with the human respiratory pathogen Haemophilus
influenzae was tested in two in vivo and one in vitro models. Conscious
vaccinated guinea pigs exposed to aerosolized histamine became asphyxial
significantly faster than saline-treated controls. Also the bronchoconstriction
in anaesthetized guinea pigs as a result of i.v. histamine was significantly potentiated
in the H. influenzae pretreated group. Isoprenaline (30 micrograms/kg)
partially inhibited the bronchoconstriction. The difference in histamine
sensitivity between the two groups however remained. Protection against
bronchoconstriction by atropine on the other hand was significantly enhanced in
the vaccinated animals. This suggests a hyperreactivity of the parasympathetic,
cholinergic pathways as a result of H. influenzae vaccination.
PMID: 6335351, UI: 85118726
Terpstra GK, Raaijmakers JA, Hamelink M, Kreukniet J. Effects of
Haemophilus influenzae vaccination on the (para-)sympathic-cyclic
nucleotide-histamine axis in rats.Ann Allergy 1979 Jan;42(1):36-40
To determine whether Haemophilus influenzae
could be a factor in human atopy its effects were studied on the
(para-)Sympathic Cyclic nucleotide-histamine axis in rats. Haemophilus
influenzae vaccination induced changes in the cholinergic system compatible
with higher cyclic GMP levels and enhanced histamine release. The authors
suggest an involvement of the cholinergic system in Haemophilus influenzae
vaccination effects. PMID: 216288, UI: 79101862
Basson E, Di Maio M, Stamm D, Cagnin S, Berger C, Floret D.
Arch Pediatr 1996 Apr;3(4):342-4 [Haemophilus influenzae meningitis following
vaccination. Consequence or coincidence]?[Article in French] Unite de
reanimation pediatrique, hopital Edouard-Herriot, Lyon, France.
BACKGROUND: The introduction of vaccines against Haemophilus influenzae type
b (Hib) has had a substantial impact on Hib infections. Their use has
established their excellent safety profiles but occasional adverse effects have
been reported.
CASE REPORT: A 4 month-old infant was admitted for a severe form of Hib
meningitis with septicemia whose first manifestations developed 3 hours after
the first immunization with a conjugate vaccine against Hib (PRP-T). The
outcome was good without any sequelae.
DISCUSSION: A dramatic decrease in serum antibodies due to antigen-antibody
reaction during the first days
after immunization has been reported; this mechanism and some epidemiological
data could favor the hypothesis that the vaccine is responsible for the
infection, at least the unconjugated vaccines.
CONCLUSION: Any fever occurring in the immediate post-immunization period must
alert the possibility of a Hib infection.
http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8762955&form=6&db=m&Dopt=b
Granoff
DM, et al.Host and bacterial factors associated with Haemophilus influenzae
type b disease in Minnesota children vaccinated with type b polysaccharide
vaccine. J Infect Dis. 1989 May;159(5):908-16. PMID: 2785147; UI:
89215362
Host and bacterial factors were evaluated
among 86 Minnesota children with Haemophilus influenzae type b disease detected
by active surveillance after introduction of type b polysaccharide vaccine in
the state. Children were 2-6 y of age. Thirty-three (38%) had been vaccinated.
There was no significant difference between the frequency of low serum
concentrations of IgM, IgA, IgG, or IgG2 in the vaccinated and nonvaccinated
subjects (13% vs. 8%, P = .5). The presence of the Gm immunoglobulin allotype
phenotype (1,3,17;23;5,13,21), previously associated with a lower relative risk
of vaccine failure in children from other states, was associated with a
fourfold decrease in the relative risk of vaccine failure in Minnesota (P less
than .07). Haemophilus isolates from 58 of the children were available for
clonal characterization by multilocus electrophoresis and outer membrane
protein subtyping. There were no significant differences between the clone
distribution of the strains causing disease in vaccinated and nonvaccinated
patients, and nearly all disease-producing clones in Minnesota also are known
to cause disease in other areas of the country. Thus, vaccine failure in
Minnesota is infrequently associated with hypogammaglobulinemia or with
infection by unusual clones of a H. influenzae type b. Also, the Gm phenotype
associated with protection against vaccine failure in other areas of the USA
appears to be protective in Minnesota. PMID: 2785147, UI: 89215362
Granoff et al. (1986) analysed 228 reports of invasive disease due to Hib in
vaccinated children submitted to the FDA administration between May 1985 and
September 1987. Over 90% of these children were more than 24 months of age,
when the vaccine is supposed to be somewhat effective
Granoff et al. (1986)] deals with 55 cases of invasive Hib diseases
occurring in Children at least three weeks after vaccination. Meningitis
developed in 39 children of whom 3 died and 6 had neurologic after-effects. The
level of antibody to Hib in convalescent-phase serum from 31 of the vaccinated
children who had Hib disease was significantly lower than that in the serum
from 25 patients of similar age (range 17 to 47 months) with the disease who
had never received the Hib vaccine.
"Risk factors for invasive Haemophilus influenzae disease among
children 2-16 years of age in the vaccine era, Switzerland, 1991-1993"
(International Journal of Epidemiology, vol. 25, no. 6, December 1996, pp.
1280-5): "Continued surveillance, and detailed investigation of direct and
indirect effects of conjugated vaccines and risk factors…are important."
143 cases with invasive disease were selected, and vaccination status
ascertained. "Cases more often than controls reported suffering from
asthma and allergies… The observed association between asthma and epiglottitis
is novel and deserves further investigation."
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