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“Healing Autism: No Finer a Cause on the Planet”

January 23, 2002        News Morgue Search  www.feat.org/search/news.asp

RESEARCH

·        M.I.N.D. Institute Begin Work With Families On California Autism Study

·        Functional Neuroimaging And Childhood Autism

·        Simon Baron-Cohen Responds to Criticism over Asperger

Conjecture

·        Reader’s Posts

 

 

M.I.N.D. Institute Begin Work With Families On California Autism Study

Parents of 1,000 California children enrolled in the state’s developmental-disabilities system are receiving letters in the mail asking them to participate in a study examining an unprecedented increase in autism in the state.

The M.I.N.D. Institute at UC Davis is conducting the study to help explain the reasons behind a 273 percent increase in the number of autistic children entering the state’s 21 regional centers between 1987 to 1998. The upsurge was highlighted in a 1999 report by the California Department of Developmental Services (DDS), which operates the centers. As a result of that report, the state Legislature and Gov. Gray Davis directed DDS and the M.I.N.D. Institute to identify factors that may be responsible for the increase, funding the effort with a $1 million appropriation.

The latest DDS figures available show that 2,331 persons entered the regional center system with a diagnosis of autism during 2001. That amounts to an average of six new individuals a day.

Opinions vary among researchers and parents as to the reasons behind the increase, said UC Davis pediatric epidemiologist Robert S. Byrd, who is the principal investigator on the study. Byrd is asking parents to describe what they think caused their child’s disorder as part of the study.

“In this study, we are examining a number of possibilities, including whether there is an actual increase, whether the criteria used to diagnose autism has changed over time or if by applying today’s diagnostic standards, children have been misclassified,” Byrd said. “We also are determining whether the number of families with autistic children moving into California accounts for some of the observed increase in autism cases.”

Byrd said his team is enrolling 1,000 children and their families—

500 children diagnosed with autism and 500 diagnosed with other developmental disabilities who have been randomly selected from the DDS regional centers—to participate in the study. The two groups of children are divided into two age groups, based on year of birth. The first group was born between 1983 to 1985 (15 to 17 years old) and the second between 1993 to 1995 (5 to 7 years old).

As part of the study, researchers are recording if and how the characteristics of children reported with autism in California have changed over time. They also are asking about specific medical or developmental problems, such as mental retardation, allergies, gastrointestinal disorders, pregnancy and birth complications.

“We are also asking about regression, that is whether children seemed normal at birth and had relatively normal development to a certain point, then lost the ability to do things that other children who are developing normally might be expected to do,” Byrd said. “Parents and caregivers frequently report these problems, which have not been systematically studied. Our research should be able to estimate how much these occur and whether they have changed over the years between the two age groups we are evaluating.”

As part of the study, the researchers are reviewing the children’s regional center records. All participating families are completing a questionnaire while families of autistic children are taking part in an autism diagnostic interview at their regional center.

Results of the statewide study will be compiled and presented to the California Legislature in early summer.

Established in 1998 through the efforts of four Sacramento families whose children have autism, the M.I.N.D. Institute at UC Davis is a unique multidisciplinary research institute devoted to the study of autism and other neurodevelopmental disorders.

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Functional Neuroimaging And Childhood Autism

http://link.springer.de/link/service/journals/00247/contents/01/00570/

Nathalie Boddaert1, 2,  and Monica Zilbovicius2, 3

(1)  Service de Radiologie Pédiatrique, Necker-Enfants Malades Hospital, 149 rue de Sèvres, 75015 Paris, France

(2)  Service Hospitalier Frédéric Joliot, DRM, DSV, CEA, Orsay, France

(3)  INSERM Unité 316, CHU Bretonneau, Tours, France Presented in part at 37th Annual Congress of ESPR, Lisbon, May 2000

 

Abstract. Childhood autism is now widely viewed as being of developmental neurobiological origin. Yet, localised structural and functional brain correlates of autism have to be established. Structural brain-imaging studies performed in autistic patients have reported abnormalities such as increased total brain volume and cerebellar abnormalities.

However, none of these abnormalities fully account for the full range of autistic symptoms. Functional brain imaging, such as positron emission tomography (PET), single photon emission computed tomography (SPECT) and functional MRI (fMRI) have added a new perspective to the study of normal and pathological brain functions.

In autism, functional studies have been performed at rest or during activation. However, first-generation functional imaging devices were not sensitive enough to detect any consistent dysfunction. Recently, with improved technology, two independent groups have reported bilateral hypoperfusion of the temporal lobes in autistic children. In addition, activation studies, using perceptive and cognitive paradigms, have shown an abnormal pattern of cortical activation in autistic patients.

These results suggest that different connections between particular cortical regions could exist in autism. The purpose of this review is to present the main results of rest and activation studies performed in autism.

© Springer-Verlag 2001

 

 

 

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Simon Baron-Cohen Responds to Criticism over Asperger Conjecture

[It started with a piece, “The Geek Disease” in a trendy pop culture magazine called Wired last December.  This straight-faced revenge of the nerds article offered, amongst other things, an anecdotal portrayal of a high concentration of Asperger Syndrome people being in the vanguard of Silicon Valley’s wealthy Dot.Com-Modes.

[Then more recently appearing in Edge online magazine and then two days ago in this newsletter, was an intended fanciful piece picking up on the theme and carrying it further to lofty conjectures about a revolutionary evolution of a new generation of genetic autistic offspring. The newfound wealth and stature of this disabled, but clever class of citizens resulted in their easier matings like never before.  Thus, almost proving once again, that love, with a little boost by wealth, conquers all. What makes this mundacious conjecturing significant, is it’s public proponent: a well-known and established, ivory-towered autism expert Simon Baron-Cohen of Cambridge.

[In a pointed essay sarcaustically entitled, “Geeks Get Lucky”, which appeared here yesterday, noted autism and vaccine reform activist Mark Blaxill took Baron-Cohen to task for his musings.

[Finally, here Simon Baron-Cohen responds to Blaxill in protracted and referenced detail. -LS]

 

First, you of course realise that neither Edge, nor Wired Magazine, are scientific journals. And that my contribution to each of these recently are not scientific contributions. I recognize that too.

In the case of Edge, the invitation was to contribute a question for the forthcoming year. Nothing more. So what I did was raise a question, and then mere speculation. I did not attempt to answer the question. I simply explored the question along the lines of “suppose this were true?”

If I were to test various answers to the question, I would design a series of experiments, which as you realise would take years and years to answer. And even then, such research could fail. We both know that.

So, such speculation should be treated as nothing more than that.  Speculation can of course give rise to new hypotheses to test, or it can be ignored. But it should certainly not be treated as established fact. The testing of these new hypotheses could of course take years, if not decades.

I don’t take Wired Magazine as reporting reliable data either, of course. The article there about Silicon Valley was mere anecdote, written by a journalist, not a scientist. And my contribution to the same issue was nothing more than a magazine interview. So please do treat the content of both of those pieces with a large dose of salt.

But of course speculation doesn’t come out of nowhere. There are some data which offers fragments of clues for the idea of a link between talent in “systemising” (such as is needed in maths, physics, engineering, computer science, music, linguistics, or history, for example) and autism. In Edge and in Wired there was no scope for presenting a bibliography of such clues, but now that you have responded, I can at least give you a handful of the references here. They are listed at the end of this document. But none of these directly addresses the speculation about the impact of the airplane and the computer. These remain mere speculations.

Much more importantly, I am very concerned that these speculations may have caused offence. As you may realize, having dedicated my life to attempting to both understand and help the situation of people with autism spectrum conditions, the last thing I want to do is cause any offence to them or their long suffering families. So, if any offence has been taken, please accept my apologies unreservedly. But I am committed to trying to understand what is causing autism, including what is causing the apparent increase in cases.

So is my question and speculation in Edge “bad science”? Well, it is not science at all, and nor was it intended to be. The science, if someone chooses to do it, is yet to be done. It is a discussion paper, and nothing more. Some of the speculation may be hard to test scientifically, but one study that needs to be done is to look not at the rate of autism among people of mixed cultural marriage (since mixed cultural marriage could occur for a whole host of reasons) but rather at the rate of mixed cultural marriage in married adults with Asperger Syndrome. This is predicted to be higher than an appropriate comparison group.

Is my speculation in Edge “malicious” ? I certainly hope not. I am one of the few academics who is frequently in the media arguing that we should celebrate autism as part of the individual differences in the population, and stress the positive qualities and not just the disabilities. I want to see a world in which people with autism can feel accepted and valued.

I should say that my team was one of the first to report, in the scientific literature, a study of the new rates of autism (Scott, F, Baron-Cohen, S, Bolton, P, & Brayne, C, (in press) Prevalence of autism spectrum conditions in children aged 5-11 years in Cambridgeshire, UK.  Autism), identifying a rate of approximately 1 in 200 children, as compared to the traditional figure of 4 in 10,000. We also took these findings to the House of Commons All Party Parliamentary Group as soon as the study was complete, so as to raise awareness among MPs of this important fact, rather than waiting for the usual time-lag in scientific journal publishing before disseminating these results. I agree that the scientific community has been far to slow to acknowledge this change in prevalence, though it takes time to carry out a study of such changes.

I think you are absolutely right that the apparent rise in rates of autism could be due to a whole host of factors, and regressive autism (which you mention) may be one of these. Like my piece in Edge, this is also speculation, and one that would be interesting to test systemically. Other possible factors of course simply include better awareness of the range of the spectrum, as I mentioned in Edge.

Regarding your remark that our attempts at developing a screen for autism at 18m old have failed, I consider that we have learnt alot from that study. For one thing, we have learnt that the absence of the key behaviours of joint attention and pretend play carry a risk for autism rate of over 80%. This is an important discovery, and means that some children with autism can be picked up on the basis of them not showing these behaviours.  But we have also learnt that the presence of these key behaviours does not mean you will not develop autism. Indeed, the screen as it stands also misses about 80% of children with autism spectrum conditions. So we are back to the drawing board, and will be spending the next years fine-tuning the screening methodology until the screen works more successfully. That is of course the nature of science.

Back to offence. I have to admit that I found the headline in Wired Magazine highly offensive, referring to autism and Asperger Syndrome as “geek syndrome”. It saddens me that you have picked up this terminology in your email too. I don’t find it useful and I think it could perpetuate discrimination which I am keen to overcome with more positive language and a more positive focus.

Equally, my speculative piece is by no means meant to be an indicator of a eugenicist or social engineering viewpoint.  It saddens me even more that you imagine my hope would be that people with the genes for autism or Asperger Syndrome should not have children. My beliefs could not be more different to these. My view is that autism spectrum conditions should be destigmatised in society, and that we should do everything we can to make it easier for people with autism spectrum conditions to have access to the same rights, opportunities and quality of life as people without autism spectrum conditions.

With best wishes

Simon Baron-Cohen

 

      Reference for some of the clues for the link between autism and

systemising skills:

1.   Baron-Cohen, S, (1997) Are children with autism superior at folk physics? In Wellman, H, & Inagaki, K, (eds) The emergence of core domains of thought: Children¹s reasoning about physical, psychological and biological phenomena.  New Direction for Child Development Series, 75, 45-54.Jossey-Bass Inc.

2.   Baron-Cohen, S, Wheelwright, S, Stott, C, Bolton, P, & Goodyer, I,

(1997)        Is there a link between engineering and autism? Autism, 1, 101-108.

3.   Jolliffe, T, & Baron-Cohen, S, (1997)  Are people with autism or

Asperger Syndrome faster than normal on the Embedded Figures Task? Journal

of Child Psychology and Psychiatry, 38, 527-534

4.   Baron-Cohen, S, Bolton, P, Wheelwright, S, Scahill, V, Short, L,

Mead, G, & Smith, A, (1998) Does autism occur more often in families of physicists, engineers, and mathematicians?  Autism, 2, 296-301.

5.   Baron-Cohen, S, Wheelwright, S, Stott, C, Bolton, P, & Goodyer, I,

(1998)        Engineering and autism: exploring the link further: a reply to

Wolff, Brausberg and Islam.  Autism, 2, 98-104.

6.   Plaisted, K, O¹Riordan, M, & Baron-Cohen, S, (1998) Enhanced visual search for a conjunctive target in autism: A research note.  Journal of Child Psychology and Psychiatry, 39, 777-783.

7.   Baron-Cohen, S, Wheelwright, S, Stone, V, & Rutherford, M, (1999) A mathematician, a physicist, and a computer scientist with Asperger Syndrome: performance on folk psychology and folk physics tests. Neurocase, 5, 475-483.

8.   Baron-Cohen, S, & Wheelwright, S, (2001) The Autism-Spectrum

Quotient: Evidence from Asperger Syndrome/high-functioning autism, males and females, scientists, and mathematicians.  Journal of Autism and Developmental Disorders, 31, 5-17.

9.   Baron-Cohen, S, Wheelwright, S, Scahill, V, Spong, A, & Lawson, J,

(2001) Are intuitive physics and intuitive psychology independent? A test

with children with Asperger Syndrome.  Journal of Developmental and Learning

Disorders. ------------- Simon Baron-Cohen Professor of Developmental

Psychopathology Autism Research Centre University of Cambridge Departments

of Experimental Psychology and Psychiatry Downing St Cambridge CB3 9JL

www.psychiatry.cam.ac.uk/arc

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Reader’s Posts

Family looking to move to MD , or bordering areas of VA or PA.  Looking to get information on schools, services and medicaid/waivers.  Also looking to get different experiences from different counties, positive and negative.

Our son is 4.5 and nonverbal. Rolex20032@aol.com

Calgary, Alberta.  Needed for four hours Sundays, individual to help HFA ten

year old with homework and community trips.  Would suit rehab or education

student.  Miamishooves@hotmail.com

I am relocating from New Mexico to Smithville, Texas, this summer. Anyone

with information of available services for a profoundly autistic 12 year

old, (city or statewide), would be greatly appreciated. I am only familiar

with services that New Mexico offers.  witsiepe@earthlink.net

Looking for videos on ABA / Discrete Trial Teaching to use to work with my

son (3 ½) who is PDD.  I would love to be able to borrow - but would also

like a list of the best available.  I live in the Chillicothe, Ohio area and

there are very little resources. Wendy Fout toddwendynoah@bright.net

San Juan Capistrano, CA Family is looking for weekend tutor to join home

program.  8 year old nonverbal boy.  Work on play therapy, self help and

community outings.  Holidays/summers extra hours available.  Great program.

email: DAWCFP@aol.com FAX: 949-489-8377

Looking for a lawyer who is experienced with the new suits being filed for autism believed to be related to the MMR shot and don’t know where to turn.

I have read my sons medical records and strongly believe these two things

are related to one another. Alice Fitzmorris alicedj@netzero.net

We are seeking special ed info in Caloosa County, CA.  We have a 7 ½ year

old Autistic/Downs child who is non verbal. The school in Three Rivers,

CA/Tulare county have taken us to fair hearing for placement outside LEA

with a 1+ hr bus ride.  We are looking at alternatives in case we lose this

case.  conev@thegrid.net

 

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