http://www.asmusa.org/pcsrc/40icaac/23115.htm

40th ICAAC

Interscience Conference on Antimicrobial Agents and Chemotherapy A meeting of the American Society for Microbiology September 17-20, 2000, Toronto, Ontario, Canada For more information on any presentation at the 40th ICAAC contact Jim Sliwa, ASM Communications at jsliwa@asmusa.org.

 

Evidence to suggest that we should be investigating the inclusion of strains currently causing infection in pertussis vaccine formulations (Session 6, Paper 61)

David Farrell

Queensland Health Pathology Service

+61 7 46316562

djfarrell@bigpond.com

Results and conclusions

Sequencing of several B. pertussis genes important for infection revealed differences in Australian strains currently causing infection (contemporary strains) compared to strains used in vaccine manufacture. Interestingly, these differences were identical to those described in previous work performed by researchers in the Netherlands. In a mouse respiratory infection model, contemporary strains were found to cause greater lung damage than vaccine strains. Conversely, tracheal colonization and inflammation was lower for contemporary strains.  After vaccination with current vaccine strains, challenge with a contemporary strain resulted in a lower cell-mediated immune response (thought to be important for long-term protection) than when the vaccine was made from the contemporary strain. In addition, in mice vaccinated with a vaccine strain and challenged with a contemporary strain, a markedly increased time to clear bacteria from the lung was observed when compared to challenge with the vaccine strain (6 weeks compared to 1 week). These results suggest that an increase in time to bacterial clearance is occurring in mice vaccinated with current vaccine strains and challenged with contemporary strains. Contemporary strains were found to have undetectable levels of filamentous haemagglutin, a protein that is important for colonization of the upper respiratory tract. A limitation of this study is the small number of strains tested. However, the data obtained would have major implications for the selection of vaccine components. Therefore, a larger and geographically diverse study is needed to confirm this preliminary data.

Further information

Despite successful vaccination programs in developed countries, infections caused by Bordetella pertussis result in significant mortality and morbidity in humans. Hundreds of thousands of deaths, the majority in developing countries, are attributed to pertussis annually. In addition, adult pertussis is becoming increasingly recognized as a disease of chronic cough in adolescents and the elderly. Although pertussis in adults is not life threatening, it is an important cause of morbidity, economic loss due to decreased productivity, and as a reservoir for transmission to susceptible individuals.

Immunity from vaccination is not as complete as that obtained from natural infection and tends to wane in teenage years. Vaccine efficacy is measured by both the immunoglobulin response and the mouse intracerebral challenge test, neither of which assess long-term immunity. Whole cell and acellular vaccines have been shown to be efficacious with regards to both of these parameters. Clinical trials have failed to show a clear correlation between serum antibody levels and protection. Cell-mediated immunity (CMI) has been shown important in providing long-term immunity to pertussis. There is conflicting evidence with regards to the ability of acellular component vaccines to generate a significant CMI response, but data to date suggests that the response is not as efficient as that generated with the whole cell vaccines.

Changes in the genetic sequence of virulence factors used in for vaccine production have recently been described (Mooi et al., 1998). It is thought that these changes may be an evolutionary survival tactic by the organism in response to vaccination. If contemporary strains of B. pertussis differ from strains used in the production of vaccines in a significant way (i.e. in the pathogenesis of and/or the immune response to infection) then this phenomenon would have major implications for component selection in vaccine formulations.

The present study suggests that contemporary strains are genetically different from vaccines (confirming the previous study in the Netherlands) and that these differences are present in strains from geographically diverse sites. The study also suggests that contemporary strains produce a different disease pattern than vaccine strains and produce an altered immune response in the host. Although pertussis vaccination has saved millions of lives, the organism has not been eradicated. The present study may provide a clue to the reasons why – if the organism has adapted to persist longer and cause less damage to a previously immunized host, it is then available to be transmitted to a non-immunized host. Inclusion of contemporary strains in current vaccines may provide an immune response that stops carriage of the organism, diminishes the reservoir and finally eradicates the disease.

Reference

Mooi, F.R., van Oirschot, H., Heuvelman, K., van der Heide, H.G.J., Gaastra, W. & Willems, R.J.L. 1998, ‘Polymorphism in the Bordetella pertussis virulence factors P.69/pertactin and pertussis toxin in the Netherlands: temporal trends and evidence for vaccine-driven evolution’, Infection and Immunity, vol.66, no.2, pp.670-675.

 

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