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Antibody Theory
Quotes Disease theory

Antibodies used as measure of immunity:
"He said the normal trials on a new vaccine were not possible in Britain because of the relatively small numbers of people who contracted the disease. Instead scientists had tested whether the vaccine produced sufficient antibodies."--Media report on meningitis C vaccine

Antibodies not a measure of immunity:
"Human trials generally correlate "antibody" responses with protection - that is if the body produces antibodies (proteins) which bind to vaccine components, then it must be working and safe. Yet Dr March says antibody response is generally a poor measure of protection and no indicator at all of safety. "Particularly for viral diseases, the 'cellular' immune response is all important, and antibody levels and protection are totally unconnected."--Private Eye 24/1/2002

"The fallacy of this (antibody theory) was exposed nearly 50 years ago, which is hardly recent. A report published by the Medical Research Council entitled 'A study of diphtheria in two areas of Gt. Britain, Special report series 272, HMSO 1950 demonstrated that many of the diphtheria patients had high levels of circulating antibodies, whereas many of the contacts who remained perfectly well had low antibody."--Magda Taylor, Informed Parent

"Just because you give somebody a vaccine, and perhaps get an antibody reaction, doesn’t mean a thing. The only true antibodies, of course, are those you get naturally. What we’re doing [when we inject vaccines] is interfering with a very delicate mechanism that does its own thing. If nutrition is correct, it does it in the right way. Now if you insult a person in this way and try to trigger off something that nature looks after, you’re asking for all sorts of trouble, and we don’t believe it works."—Glen Dettman Ph.D, interviewed by Jay Patrick, and quoted in "The Great American Deception," Let’s Live, December 1976, p. 57.

"Many measles vaccine efficacy studies relate to their ability to stimulate an antibody response, (sero-conversion or sero-response). An antibody response does not necessarily equate to immunity......... the level of antibody needed for effective immunity is different in each individual.....immunity can be demonstrated in individuals with a low or no detectable levels of antibody.    Similarly in other individuals with higher levels of antibody there may be no immunity. We therefore need to stay clear on the issue: How do we know if the vaccine is effective for a particular individual when we do not know what level of antibody production equals immunity?"--Trevor Gunn BSc

A jab in the dark

" The antibody business: Millions of screening tests are distributed, each blood sample needs to be tested (4 millions in Germany alone)  ... The therapy business: Antiviral medication, 3 or 4 or 5 fold combinations, AIDS can´t be topped in this department. ....... With intoxication hypotheses on the other hand you cannot make any money at all. The simple message is: Avoid the poison and you won´t get sick. Such hypotheses are counterproductive insofar as the toxins (drugs, alcohol, pills, phosmet) bring high revenues. The conflict of interests is not resolvable: What virologist who does directly profit millions from their patent rights of the HIV or HCV tests (Montagnier, Simon Wain-Hobsen, Robin Weiss, Robert Gallo) can risk to take even one look in the other direction."--By Claus Köhnlein

"When they say immunogenicity what they actually mean is antibody levels. Antibody levels are not the same as IMMUNITY. The recent MUMPS vaccine fisaco in Switzerland has re-emphasised this point. Three mumps vaccines—Rubini, Jeryl-Lynn and Urabe (the one we withdrew because it caused encepahlitis) all produced excellent antibody levels but those vaccinated with the Rubini strain had the same attack rate as those not vaccinated at all (12), there were some who said that it actually caused outbreaks."--Dr Jayne Donegan

"Whenever we read vaccine papers the MD researchers always assume that if there are high antibody levels after vaccination, then there is immunity (immunogencity). But are antibody levels and immunity the same?  No! Antibody levels are not the same as IMMUNITY. The recent MUMPS vaccine fiasco in Switzerland has re-emphasized this point. Three mumps vaccines-Rubini, Jeryl-Lynn and Urabe (the one withdrawn because it caused encephalitis) all produced excellent antibody levels but those vaccinated with the Rubini strain had the same attack rate as those not vaccinated at all, there were some who said that it actually caused outbreaks. Ref: Schegal M et al Comparative efficacy of three mumps vaccines during disease outbreak in Switzerland: cohort study. BMJ, 1999; 319:352-3."--Ted Koren DC

"In order to better grasp the issue of vaccine effectiveness, it would prove helpful for us to go back to the early theoretical foundation upon which current vaccination and disease theories originated. In simplest terms, the theory of artificial immunization postulates that by giving a person a mild form of a disease, via the use of specific foreign proteins, attenuated viruses, etc., the body will react by producing a lasting protective response e.g., antibodies, to protect the body if or when the real disease comes along.
        This primal theory of disease prevention originated by Paul Ehrlich--from the time of its inception--has been subject to increasing abandonment by scientists of no small stature. For example not long after the Ehrlich theory came into vogue, W.H. Manwaring, then Professor of Bacteriology and Experimental Pathology at Leland Stanford University observed:
I believe that there is hardly an element of truth in a single one of the basic hypothesis embodied in this theory. My conviction that there was something radically wrong with it arose from a consideration of the almost universal failure of therapeutic methods based on it . . . Twelve years of study with immuno-physical tests have yielded a mass of experimental evidence contrary to, and irreconcilable with the Ehrlich theory, and have convinced me that his conception of the origin, nature, and physiological role of the specific 'antibodies' is erroneous.33
        To afford us with a continuing historical perspective of events since Manwaring's time, we can next turn to the classic work on auto-immunity and disease by Sir MacFarlane Burnett, which indicates that since the middle of this century the place of antibodies at the centre stage of immunity to disease has undergone "a striking demotion." For example, it had become well known that children with agammaglobulinaemia--who consequently have no capacity to produce antibody--after contracting measles, (or other zymotic diseases) nonetheless recover with long-lasting immunity. In his view it was clear "that a variety of other immunological mechanisms are functioning effectively without benefit of actively produced antibody."34
        The kind of research which led to this a broader perspective on the body's immunological mechanisms included a mid-century British investigation on the relationship of the incidence of diphtheria to the presence of antibodies. The study concluded that there was no observable correlation between the antibody count and the incidence of the disease." "The researchers found people who were highly resistant with extremely low antibody count, and people who developed the disease who had high antibody counts.35 (According to Don de Savingy of IDRC, the significance of the role of multiple immunological factors and mechanisms has gained wide recognition in scientific thinking. [For example, it is now generally held that vaccines operate by stimulating non-humeral mechanisms, with antibody serving only as an indicator that a vaccine was given, or that a person was exposed to a particular infectious agent.])
        In the early 70's we find an article in the Australian Journal of Medical Technology by medical virologist B. Allen (of the Australian Laboratory of Microbiology and Pathology, Brisbane) which reported that although a group of recruits were immunized for Rubella, and uniformly demonstrated antibodies, 80 percent of the recruits contracted the disease when later exposed to it. Similar results were demonstrated in a consecutive study conducted at an institution for the mentally disabled. Allen--in commenting on herb research at a University of Melbourne seminar--stated that "one must wonder whether the . . . decision to rely on herd immunity might not have to be rethought.36
        As we proceed to the early 80s, we find that upon investigating unexpected and unexplainable outbreaks of acute infection among "immunized" persons, mainstream scientists have begun to seriously question whether their understanding of what constitutes reliable immunity is in fact valid. For example, a team of scientist writing in the New England Journal of Medicine provide evidence for the position that immunityto disease is a broader bio-ecological question then the factors of artificial immunization or serology. They summarily concluded: "It is important to stress that immunity (or its absence) cannot be determined reliable on the basis of history of the disease, history of immunization, or even history of prior serologic
determination.37
        Despite these significant shifts in scientific thinking, there has unfortunately been little actual progress made in terms of undertaking systematically broad research on the multiple factors which undergird human immunity to disease, and in turn building a system of prevention that is squarely based upon such findings. It seems ironic that as late as 1988 James must still raise the following basic questions. "Why doesn't medical research focus on what factors in our environment and in our lives weaken the immunesystem? Is this too simple? too ordinary? too undramatic? Or does it threaten too many vested interests . .
?" 38"---Dr Obomsawin MD

"FROM REPEATED medical investigations, it would seem that antibodies are about as useful as a black eye in protecting the victim from further attacks. The word "antibody" covers a number of even less intelligible words, quaint relics of Erlich’s side-chain theory, which the greatest of experts, McDonagh, tells us is "essentially unintelligible". Now that the old history, mythology and statistics of vaccination have been exploded by experience, the business has to depend more upon verbal dust thrown in the face of the lay public. The mere layman, assailed by antibodies, receptors, haptophores, etc., is only too pleased to give up the fight and leave everything to the experts. This is just what they want, especially when he is so pleased that he also leaves them lots and lots of real money.
    The whole subject of immunity and antibodies is, however, so extremely complex and difficult, especially to the real experts, that it is a relief to be told that the gaps in their knowledge of such things are still enormous.
    We can obtain some idea of the complexity of the subject from The Integrity of the Human Body, by Sir Macfarlane Burnet. He calls attention to the fact—the mystery—that some children can never develop any antibodies at all, but can nevertheless go through a typical attack of, say, measles, make a normal recovery and show the normal continuing resistance to reinfection. Furthermore, we have heard for years past of attempts made to relate the amount of antibody in patients to their degree of immunity to infection. The, results have often been so farcically chaotic, so entirely unlike what was expected, that the scandal has had to be hushed up—or put into a report, which is much the same thing (vide M.R.C. Report, No. 272, May 1950, A Study of Diphtheria in Two Areas of Great Britain, now out of print). The worse scandal, however, is that the radio is still telling the schools that the purpose of vaccinating is to produce antibodies. The purpose of vaccinating is to make money!"---Lionel Dole

Crone, NE; Reder, AT; Severe tetanus in immunized patients with high anti-tetanus titers; Neurology 1992; 42:761-764;
Article abstract: Severe (grade III) tetanus occurred in three immunized patients who had high serum levels of anti-tetanus antibody. The disease was fatal in one patient. One patient had been hyperimmunized to produce commercial tetanus immune globulin. Two patients had received immunizations one year before presentation. Anti-tetanus antibody titers on admission were 25 IU/ml to 0.15 IU/ml by hemagglutination and ELISA assays; greater than 0.01 IU/ml is considered protective. Even though one patient had seemingly adequate anti-tetanus titers by in vitro measurement 0.20 IU in vivo mouse protection bioassays showed a titer less than 0.01 IU/ml, implying that there may have been a hole in her immune repertoire to tetanus neurotoxin but not to toxoid. This is the first report of grade III tetanus with protective levels of antibody in the United States. The diagnosis of tetanus, nevertheless, should not be discarded solely on the basis of seemingly protective anti-tetanus titers. http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1565228&form=6&db=m&Dopt=b

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